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Essay / Transmission of pain signals from the brain to the spine. Pain was defined by Coates & Hindle as an unpleasant emotional and sensory experience that signals potential or actual tissue damage (2011, p. 213). Pain is a common human experience and can result from injury or illness. There are two main types of pain: acute pain is short-lived, lasting a few minutes or several days, and its onset is often rapid. It results from the activation of painful nerve endings or nociceptors by internal or external painful stimuli. On the other hand, chronic pain is continuous and sometimes recurring and can last for weeks, months, or even years. Chronic pain is generally not localized or related to the tissue experiencing trauma (Draper & Knight, 2007, p. 104). Various theories have been proposed to explain the mechanism underlying pain transmission and perception. These include the specificity theory which holds that specific fibers and pain receptors are activated by injury, after which pain signals are projected via the spinal pathway to an area of pain. brain that interprets pain. In this regard, specificity theory virtually equates peripheral injury with the psychological experience caused by pain (Anderson, 2004, p. 355). However, this theory has several limitations, as research on pain has intensified over time. In light of this, the gate theory proposed by Melzack and Wall has made a major contribution to the understanding of pain transmission and perception (Pain Game Part 2, 2011). Research has demonstrated that pain is affected by psychological and physiological factors, which helps explain the mechanism underlying pain inhibition and/or facilitation.... middle of article.. ....it has been noted that the gate control theory proposed by Melzack and Wall in 1965 formed the basis for understanding the process of transmission of pain signals. The dorsal horn of the spinal cord is the region of the CNS that controls the passage of pain signals by opening and/or closing the gate. Pain can only be felt if it reaches the brain. Events that cause arousal, such as pain signals and the release of excitatory or facilitatory chemicals, cause the gate to open, while inhibitory events, such as competing nerve impulses caused by friction, trigger the gate. closing the door. The gate may also be closed due to descending inhibition reinforced by relaxation or the use of painkillers such as morphine. The brainstem is responsible for controlling the transmission of pain signals via the ascending and descending pain pathways..
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